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Anemia in sentence

55 examples of Anemia in a sentence

Even in a mild case, the result can be fever, chills, flu-like symptoms, and anemia – especially dangerous for pregnant women and young children.
Identification of genetic molecular markers for improved tea harvests in Kenya, ongoing efforts to examine alternative treatments for river blindness in Uganda, research on sickle-cell anemia in Ghana, and detailed assessments in Madagascar of the effectiveness of medicinal plants treating diabetes are examples of science-based initiatives that deserve recognition within the larger scientific community and among the public.
The world is plagued with hereditary diseases that cause very real misery: sickle cell anemia, hemophilia, type 1 diabetes, cystic fibrosis, mitochondrial diseases, polycystic kidney disease, Tay-Sachs disease, Canavan disease, mucopolysaccharidoses, some forms of breast, prostate, and colon cancer – the list goes on.
For example, scientists have known for decades that certain drugs can cause severe and precipitous anemia in people with a genetic deficiency of the enzyme G6PD.
However, this product did nothing to lessen the severity or duration of the condition, which over time leads to malnutrition, anemia, and other chronic health risks.
This concept is not new: it has been known for decades, for example, that people who have a genetic deficiency of an enzyme called G6PD can experience severe and precipitous anemia if they are exposed to certain drugs.
Now, the thalidomide family of drugs is prescribed for multiple myeloma, mantle cell lymphoma, certain skin diseases, and some forms of anemia.
Right now, a staggering 1.6 billion people worldwide suffer from anemia, a condition strongly connected to iron deficiency – and one of the symptoms of a global nutrition crisis that disproportionally affects women.
In 2012, the World Health Assembly endorsed a target to reduce the rate of anemia by 50% by 2025.
But one facet of malnutrition – and a major killer of women – is often ignored: anemia.
It is caused by a deficiency in essential nutrients, most often iron, but also folic acid, vitamin B12, or vitamin A.Although anemia can affect anyone, the majority of those affected are women of child-bearing age: in 2011, 29% of non-pregnant women worldwide (496 million people) and 38% of pregnant women (32.4 million people) aged 15-49 were anemic.
Iron-deficiency anemia alone – representing about half of all cases – causes about a million deaths per year, some three-quarters of which occur in Africa and Southeast Asia.
Moreover, anemia contributes significantly to maternal and perinatal mortality: in Asia, it is the second leading cause of maternal death.
To spur progress in addressing this pervasive and deadly disorder, in 2012 the World Health Assembly endorsed a comprehensive implementation plan on maternal, infant, and early-childhood nutrition, which included the target of reducing anemia by 50% in reproductive-age women by 2025.
But employers – especially those that provide lunch to workers – have the power to boost micronutrient consumption significantly among their labor forces, thereby reducing the incidence of anemia.
We then tested these solutions over a ten-month period on 1,310 women aged 18-42 working in four ready-made garment factories (eight in ten female garment workers in Bangladesh suffer from anemia.)
The first set of women received a nutritionally enhanced lunch (with fortified rice) every day, one IFA supplement each week, and an “enhanced” BCC module (including information on anemia and dietary diversity) each month.
The second set received their usual lunch, no supplements, and a “regular” BCC module (including information about nutrition, but not specifically about anemia or dietary diversity).
The implementation of a full nutrition package in the first factory reduced the prevalence of anemia by 32%.
This reinforces the findings of separate studies, in which fortified foods caused the anemia rate among participants to drop by 46% over 6-11 months.
Intermittent doses of IFA supplements reduced the prevalence of anemia in menstruating women by 27%.
Among the second set of women, who received the regular BCC module and no other interventions, anemia actually increased by 6%.
If the clinical evidence is not enough to convince companies to invest in combating anemia among their workers, perhaps the productivity benefits will be.
Such initiatives can serve as a model for other developing countries, where anemia continues to damage – and even end – women’s lives.
Sickle cell anemia, Ashkenazi genetic diseases, and cystic fibrosis are just three examples of the many diseases with population-specific correlations.
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